🔑 Key Definitions
Acetylcholine (ACh): Neurotransmitter at neuromuscular junction and in brain; involved in muscle control, arousal, and memory
Glutamic Acid (Glutamate): Primary excitatory neurotransmitter in CNS; involved in learning and plasticity
GABA (Gamma-Aminobutyric Acid): Primary inhibitory neurotransmitter in CNS; 10-15 times more abundant than monoamines
Alzheimer's Disease: Neurodegenerative disorder with loss of cholinergic neurons; memory and cognitive decline
Glutamic Acid (Glutamate): Primary excitatory neurotransmitter in CNS; involved in learning and plasticity
GABA (Gamma-Aminobutyric Acid): Primary inhibitory neurotransmitter in CNS; 10-15 times more abundant than monoamines
Alzheimer's Disease: Neurodegenerative disorder with loss of cholinergic neurons; memory and cognitive decline
🧠 Acetylcholine (ACh)
- Neuromuscular Junction: ACh is excitatory at skeletal muscles; causes contraction
- Cardiac Muscle: ACh is inhibitory at heart; slows heart rate (vagus nerve)
- Arousal: Cholinergic neurons in basal forebrain promote cortical activation and wakefulness
- Memory: ACh critical for attention and memory formation
- Alzheimer's Disease: Degeneration of cholinergic neurons in basal forebrain; treated with acetylcholinesterase inhibitors
🧠 Glutamic Acid (Glutamate)
- Primary Excitatory NT: Most abundant excitatory neurotransmitter in brain
- Learning & Plasticity: Critical for LTP (long-term potentiation) and memory formation
- NMDA Receptors: Glutamate receptor type crucial for synaptic plasticity
- Excitotoxicity: Excessive glutamate can kill neurons (stroke, trauma)
🧠 GABA (Gamma-Aminobutyric Acid)
- Primary Inhibitory NT: Main inhibitory neurotransmitter throughout brain
- Abundance: 10-15 times more GABA neurons than monoamine neurons
- Anxiety: GABAergic drugs (benzodiazepines) reduce anxiety
- Balance: GABA balances glutamate's excitation; prevents overexcitation
💡 Exam Tips
- ACh has OPPOSITE effects: excitatory at muscles, inhibitory at heart
- Alzheimer's = loss of ACh neurons; treat by blocking acetylcholinesterase
- Glutamate = THE excitatory NT (not just "an" excitatory NT)
- GABA = THE inhibitory NT; 10-15x more abundant than monoamines
- Remember balance: Glutamate (excites) vs GABA (inhibits)
- GABA drugs (benzodiazepines like Valium) treat anxiety by increasing inhibition